Specific changes in the respiratory apparatus in MM can be divided into the following options.
1. Diffuse myelocytic cell infiltration of the lung tissue (pulmonary plasmacytosis) or the formation of individual tumor nodules in the lung tissue .
2. The lesion of the bronchi is usually combined with the lesion of the lung tissue. Lesions are revealed along the course of the large bronchi, in the peribronchial spaces ah, under the bronchial mucosa . A tumor is protruding into the lumen of the bronchus, which has grown through the wall of the bronchus, moving apart normal tissue structures and spreading further to the lung tissue along the bronchioles and between the alveolar septa .
3. Pleurisy with MM are rare . Ss Bessmaltsev and K.M. Abdulkadyrov (2004) described a case of myeloma lesion of the lung with the development of exudative pleurisy in a patient with diclonous (IgG, IgA) myeloma. Pleural sheaths in MM are rarely affected . Clinically, myeloma pleurisy is characterized by unilateral or bilateral hemorrhagic effusion. Cytological examination revealed a large number of atypical plasma cells . Biochemical and electrophoretic examination of the pleural fluid sometimes makes it possible to detect paraprotein in it .
4. Tumors of the ribot, for the second time affecting the respiratory apparatus or squeezing the corresponding zones of the lung tissue, can come from any rib, usually destroying it, while they squeeze the adjacent zones of the lung tissue and create an x-ray effect of the “ lung tumor” . Vidal et. al. (1969) described a tumor weighing 550 g emanating from the rib and accompanied by pleurisy. The destruction of the ribs in MM takes one of the first places among the tumor lesions of the chest cell .
5. plasmacytoma upper airway can was placed a throat, larynx, nasopharyngeal space, trachea, interfere with normal breathing and ventilation break lѐ soft .
6. X-ray examination of the lungs in MM patients reveals a strengthening and deformation of the pulmonary pattern according to the interstitial type, which is caused by stagnation in small vessels with the development of pneumosclerosis, as the blood flow in the pulmonary capillaries slows down due to the increased viscosity of the plasma .